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Whether you are a parent, coach, school professional, or health care provider, this site will help you recognize, respond to, and minimize the risk of concussion or. In Pittsburgh, accomplished pathologist Dr. Bennet Omalu uncovers the truth about brain damage in football players who suffer repeated concussions in the course of. I don’t think there’s anybody as happy as the rally fans who help dig out a car from the woods about a minute into this supercut of flat-out ridiculousness, either.

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  • The most common and least serious type of traumatic brain injury is called a concussion. The word comes from the Latin concutere, which means "to shake violently.

Second- impact syndrome - Wikipedia. Second- impact syndrome (SIS) occurs when the brain swells rapidly, and catastrophically, after a person suffers a second concussion before symptoms from an earlier one have subsided. This second blow may occur minutes, days or weeks after an initial concussion. The cause of SIS is uncertain, but it is thought that the brain's arterioles lose their ability to regulate their diameter, and therefore lose control over cerebral blood flow, causing massive cerebral edema. In order to prevent SIS, guidelines have been established to prohibit athletes from returning to a game prematurely.

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For example, professionals recommend that athletes not return to play before symptoms of an initial head injury have resolved. However, the syndrome is recognized by physicians. Thus, people who participate in sports such as boxing, soccer, American football, baseball, rugby, basketball, ice hockey, pro wrestling, and skiing (especially Alpine) are at increased risk. All documented cases occurred in people younger than 2. Cingulate herniation - The brain squeezes under the falx cerebri. Transcalvarial herniation - through a skull fracture 5) Upward herniation of the cerebellum 6) Tonsillar herniation - the cerebellar tonsils herniate through the foramen magnum. Concussion temporarily changes the brain's function.

Thus athletes are advised to use protective gear such as helmets. Thus athletic trainers may prohibit return to play for any athlete who has suffered a concussion, regardless of the speed with which the symptoms reportedly resolve.

These exist in part to prevent the player from developing SIS. Every return- to- play guideline recommends that an athlete not return to competition until all concussion symptoms are absent during both rest and exercise. Saunders described the death of a football player who had died after a second, unremarkable concussion.

Kelly and others reported another football death after repeated concussions. After undergoing a head- to- head hit at football practice, a university doctor diagnosed Plevretes with a concussion and two days later was told he was allowed to resume play. Three games after returning Plevretes sustained another concussion resulting in second- impact syndrome. He was rushed to the hospital where doctors cut the right side of his skull. Five years after the injury, he still struggles to do everyday activities.

Because of Plevretes, the NCAA revised concussion guidelines. The NCAA strongly urges schools to have a concussion management plan.

They also require that an athlete have clearance by a team doctor before being allowed to return. He shrugged off the hit and went back in the game a few plays later. At the end of the game, he collapsed on the field. Lystedt had second- impact syndrome. He was airlifted to a hospital where surgeons cut both sides of his skull so that the blood clot could be taken out. He was in the hospital for months and he still has trouble doing everyday things, such as walking. To prevent other families from going through what they went through, the Lystedt family made it their goal to make a law that athletes who are suspected of a concussion are not allowed to return to play unless he or she is cleared by a licensed physician.

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